James W. Prescott, PhD sent news that research documents that BPA, an endocrine disrupter, impairs social relations until the fourth generation. The reproductive damage of BPA has been well documented. See Peat Myers interview below. The social consequences of endocrine disruption are new. The importance of this finding and its impact on the socialization of humanity is staggering.
We all know that the environment controls and regulates gene expression even damages gene structure and function. Some 80,000 chemicals dumped into our environment most of them toxic. BPA is just one, and alone represents 7 billion pounds annually.
Gestational BPA exposure affected gene expression involved in social behavior–specifically, the first generation of mice exhibited fewer social interactions with their peers and less of a desire to spend time with adult males among juvenile males. The effects continued on into the next four generations of mice.
It’s not too surprising that BPA would do this. As we mentioned, it’s an endocrine-disrupting chemical. Steroid hormones (part of the endocrine system) regulate genes for things like vasopressin and oxytocin, which are chemicals that play a key role in expressions of social behavior. Oxytocin, for instance, helps us trust others. A chemical that disrupts that system could create changes in our social behavior.
The full article can be found in Endocrinology
Impaired Social Relations is a major contributing factor to somatosensory-affectional deprivation (SSAD). We are slowly but surely creating toxic environments that is driving us to extinction.
See The Faroes Statement: Human Health Effects of Developmental Exposure to Chemicals in Our Environment
The periods of embryonic, foetal and infant development are remarkably susceptible to environmental hazards. Toxic exposures to chemical pollutants during these windows of increased susceptibility can cause disease and disability in infants, children and across the entire span of human life. Among the effects of toxic exposures recognized in the past have been spontaneous abortion, congenital malformations, lowered birth weight and other adverse effects. These outcomes may be readily apparent. However, even subtle changes caused by chemical exposures during early development may lead to important functional deficits and increased risks of disease later in life.
James W. Prescott, Ph.D.
Over a decade ago I interviewed Peat Myers, one of THE lead researchers that documented the reproductive damage Bisphenol A (parts per billion.)
His interview explains how this takes place and redefines by a power of 10 how chemicals emulate – trigger and repress – millions of years of genetic expression.